Copper biochemistry and molecular biology. Am J Clin Nutr. 1. ![]() Original Article. Primary Prevention of Cardiovascular Disease with a Mediterranean Diet. Ramón Estruch, M.D., Ph.D., Emilio Ros, M.D., Ph.D., Jordi Salas-Salvadó. Minnesota Mom of Two Diagnosed with Cancer Hours After Husband Dies From ALS: 'I'm Not Ready to Give Into It'. Nutrient Search: Foods highest in Fructose. Better Choices for Healthy Weight Loss The Better Choices approach predicts that foods closer to the top of this list. Fructose and glucose are simultaneously absorbed in equal amounts by the intestines. Excess fructose is absorbed by a different mechanism. With the popularity of "high-protein" diets, you might be tempted to believe you simply can't overeat protein. But the truth is that consuming. To explain the observed specificity of enzymes, in 1894 Emil Fischer proposed that both the enzyme and the substrate possess specific. S- 8. 11. S. In: Shils ME, Shike M, Ross AC, Caballero B, Cousins RJ, eds. Modern Nutrition in Health and Disease. Philadelphia: Lippincott Williams & Wilkins; 2. In: Erdman JW, Macdonald IA, Zeisel SH, eds. Present Knowledge in Nutrition. Ames: Wiley- Blackwell; 2. Impact of copper limitation on expression and function of multicopper oxidases (ferroxidases). Essentiality of copper in humans. Am J Clin Nutr. 1. Suppl): 9. 52. S- 9. S. Multi- copper oxidases and human iron metabolism. Copper transport and metabolism are normal in aceruloplasminemic mice. J Biol Chem. 2. 00. Targeted gene disruption reveals an essential role for ceruloplasmin in cellular iron efflux. Proc Natl Acad Sci U S A. Aceruloplasminemia. Curr Drug Targets. Hepatic iron overload or cirrhosis may occur in acquired copper deficiency and is likely mediated by hypoceruloplasminemia. J Clin Gastroenterol. In: O'Dell BL, Sunde RA, eds. Handbook of nutritionally essential minerals. New York: Marcel Dekker, Inc; 1. Is copper an antioxidant nutrient? Crit Rev Food Sci Nutr. Mechanism of copper- activated transcription: activation of AP- 1, and the JNK/SAPK and p. Iron excess treatable by copper supplementation in acquired aceruloplasminemia: a new form of secondary human iron overload? Food and Nutrition Board, Institute of Medicine. Dietary reference intakes for vitamin A, vitamin K, boron, chromium, copper, iodine, iron, manganese, molybdenum, nickel, silicon, vanadium, and zinc. Washington, D. C.: National Academy Press; 2. Effects of zinc supplementation on plasma copper/zinc ratios, oxidative stress, and immunological status in hemodialysis patients. Int J Med Sci. 2. Effect of vitamin C on copper and iron metabolism in the guinea pig. Int J Vitam Nutr Res. Influence of ascorbic acid supplementation on copper status in young adult men. Am J Clin Nutr. 1. Effect of varying ascorbic acid intakes on copper absorption and ceruloplasmin levels of young men. Aceruloplasminemia: an inherited neurodegenerative disease with impairment of iron homeostasis. Am J Clin Nutr. 1. Suppl): 9. 72. S- 9. S. Copper deficiency alters cell bioenergetics and induces mitochondrial fusion through up- regulation of MFN2 and OPA1 in erythropoietic cells. Biochem Biophys Res Commun. Cellular copper content modulates differentiation and self- renewal in cultures of cord blood- derived CD3. Br J Haematol. 2. Update on anemia and neutropenia in copper deficiency. Curr Opin Hematol. Copper deficiency and non- accidental injury. Arch Dis Child. 1. Management of copper deficiency in cholestatic infants: review of the literature and a case series. Nutr Clin Pract. 2. Copper enzyme activities in cystic fibrosis before and after copper supplementation plus or minus zinc. Copper deficiency myeloneuropathy and pancytopenia secondary to overuse of zinc supplementation. J Neurol Neurosurg Psychiatry. Denture cream: an unusual source of excess zinc, leading to hypocupremia and neurologic disease. Relapsing hypocupraemic myelopathy requiring high- dose oral copper replacement. J Neurol Neurosurg Psychiatry. Mutation in the ATP7. A gene may not be responsible for hypocupraemia in copper deficiency myelopathy. Postgrad Med J. 2. An overview and update of ATP7. A mutations leading to Menkes disease and occipital horn syndrome. Inherited copper transport disorders: biochemical mechanisms, diagnosis, and treatment. Curr Drug Metab. 2. Ceruloplasmin and cardiovascular disease. Free Radic Biol Med. Copper supplementation of adult men: effects on blood copper enzyme activities and indicators of cardiovascular disease risk. Serum copper concentration and coronary heart disease among US adults. Am J Epidemiol. 2. Serum copper as a marker of inflammation in prediction of short term outcome in high risk patients with chronic heart failure. Int J Cardiol. 2. Zinc, copper, and magnesium and risks for all- cause, cancer, and cardiovascular mortality. Epidemiology. 2. 00. Significance of serum trace element status in patients with rheumatic heart disease: a prospective study. Biol Trace Elem Res. Considerations in the development of biomarkers of copper status. Cardiovascular disease from copper deficiency- -a history. S Suppl): 4. 89. S- 4. S. Leucocyte copper, a marker of copper body status is low in coronary artery disease. J Trace Elem Med Biol. Studies in copper status and atherosclerosis. Biochem Soc Trans. Wang XL, Adachi T, Sim AS, Wilcken DE. Plasma extracellular superoxide dismutase levels in an Australian population with coronary artery disease. Arterioscler Thromb Vasc Biol. Lack of a recommended dietary allowance for copper may be hazardous to your health. J Am Coll Nutr. 1. Effects of a diet low in copper on copper- status indicators in postmenopausal women. Am J Clin Nutr. 1. Copper supplementation effects on indicators of copper status and serum cholesterol in adult males. Biol Trace Elem Res. A randomized trial of copper supplementation effects on blood copper enzyme activities and parameters related to cardiovascular health. Copper supplementation in humans does not affect the susceptibility of low density lipoprotein to in vitro induced oxidation (FOODCUE project). Free Radic Biol Med. The effect of copper supplementation on red blood cell oxidizability and plasma antioxidants in middle- aged healthy volunteers. Free Radic Biol Med. Correlation between plasma total homocysteine and copper in patients with peripheral vascular disease. The relationship between copper, homocysteine and early vascular disease in lean women with polycystic ovary syndrome. Gynecol Endocrinol. Homocysteine and atherosclerosis. Curr Opin Lipidol. Investigation of the inhibitory effects of homocysteine and copper on nitric oxide- mediated relaxation of rat isolated aorta. Br J Pharmacol. 1. Interactive effects of homocysteine and copper on angiogenesis in porcine isolated saphenous vein. Ann Thorac Surg. 2. Copper deficiency decreases plasma homocysteine in rats. Copper chelation by tetrathiomolybdate inhibits vascular inflammation and atherosclerotic lesion development in apolipoprotein E- deficient mice. Atherosclerosis. 2. Is low copper status immunosuppressive? Pt 2): S5. 9- 6. 4. Copper and immunity. Am J Clin Nutr. 1. Suppl): 1. 06. 4S- 1. S. Phagocytosis and immunoglobulin levels in hypocupremic children. Effects of low- copper diets on human immune response. Am J Clin Nutr. 1. Copper homeostasis at the host- pathogen interface. J Biol Chem. 2. 01. Prevalence and trends in low femur bone density among older US adults: NHANES 2. NHANES III. J Bone Miner Res. Estrogen replacement and skeletal muscle: mechanisms and population health. J Appl Physiol. 2. Public Health Impact of Osteoporosis. J Gerontol A Biol Sci Med Sci. Pamidronate treatment improves bone mineral density in children with Menkes disease. J Inherit Metab Dis. Copper deficiency presenting as metabolic bone disease in extremely low birth weight, short- gut infants. Effect of dietary copper intakes on biochemical markers of bone metabolism in healthy adult males. Eur J Clin Nutr. 1. No effect of copper supplementation on biochemical markers of bone metabolism in healthy adults. No effect of copper supplementation on biochemical markers of bone metabolism in healthy young adult females despite apparently improved copper status. Eur J Clin Nutr. 2. Serum copper levels in elderly patients with femoral- neck fractures. Magnesium, zinc and copper status in osteoporotic, osteopenic and normal post- menopausal women. J Int Med Res. 2. Copper supplementation and the maintenance of bone mineral density in middle- aged women. J Trace Elem Exp Med. Spinal bone loss in postmenopausal women supplemented with calcium and trace minerals. Reported zinc, but not copper, intakes influence whole- body bone density, mineral content and T score responses to zinc and copper supplementation in healthy postmenopausal women. The effect of osteoporosis on periodontal status, alveolar bone and orthodontic tooth movement. A literature review. J Int Acad Periodontol. Tooth loss and osteoporosis: to assess the association between osteoporosis status and tooth number. E1. 0. Copper deficit as a potential pathogenic factor of reduced bone mineral density and severe tooth wear. Osteoporos Int. Excess of nonceruloplasmin serum copper in AD correlates with MMSE, CSF . Clinical utility of copper, ceruloplasmin, and metallothionein plasma determinations in human neurodegenerative patients and their first- degree relatives. Copper in Alzheimer's disease: a meta- analysis of serum, plasma, and cerebrospinal fluid studies. J Alzheimers Dis. Copper excess, zinc deficiency, and cognition loss in Alzheimer's disease. Copper phenotype in Alzheimer's disease: dissecting the pathway. Am J Neurodegener Dis. Copper hypothesis in the missing hereditability of sporadic Alzheimer's disease: ATP7. B gene as potential harbor of rare variants. J Alzheimers Dis. Trace amounts of copper in water induce . Proc Natl Acad Sci U S A. Chronic copper exposure exacerbates both amyloid and tau pathology and selectively dysregulates cdk. AD. J Neurochem. 2. Dietary copper and high saturated and trans fat intakes associated with cognitive decline. Arch Neurol. 2. 00. Effect of copper intake on CSF parameters in patients with mild Alzheimer's disease: a pilot phase 2 clinical trial. J Neural Transm. 2. Intake of copper has no effect on cognition in patients with mild Alzheimer's disease: a pilot phase 2 clinical trial. J Neural Transm. 2. Impairment of interrelated iron- and copper homeostatic mechanisms in brain contributes to the pathogenesis of neurodegenerative disorders. Front Pharmacol. Transition metal abnormalities in progressive dementias. Fe and Cu do not differ in Parkinson's disease: a replication study plus meta- analysis. Neurobiol Aging. 2. PDR for Nutritional Supplements. Montvale: Medical Economics Company, Inc; 2. Manifestations of copper excess. Am J Clin Nutr. 1. Suppl): 1. 06. 9S- 1. S. Safety guidelines for copper in water. Am J Clin Nutr. 1. Suppl): 1. 09. 8S- 1. An error occurred while setting your user cookie. Please set your. browser to accept cookies to continue. This cookie stores just a. ID; no other information is captured. Accepting the NEJM cookie is.
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